max in WT synaptoneurosomes, suggesting that Src signaling could be downregulated in KI synapses. Alternatively, our capability to rescue SERT function in KI midbrain synaptoneurosomes via the inhibition of FAK signifies elevated FAK signaling downstream of the Pro32Pro33 mutant, as confirmed by greater pFAK localization in five-HT synapses. Our in… Read More